Respiratory status
Taylor et al. (1963) questioned the safety of allowing
a placental transfusion. [1]  They cautioned that a
rapid and large transfusion might overload the
circulation of a newborn.  They claimed that theirs
was a "minority opinion" favoring early clamping,  
although by the early 1960s clamping and assigning
Apgar scores was already common practice.

Taylor et al. observed respiratory status in 182 full-
term and 340 premature infants assigned to groups
for early or delayed cord clamping.  The cord was
clamped within one minute in the early-clamped
group, and to minimize placental transfusion the
infant was held above the perineum after delivery.  In
the late-clamped group the cord was clamped
between 1 and 3 minutes and to maximize placental
transfusion the infant was held below the perineum.  
A full 3 minutes was allowed to elapse before
clamping for most of the late-clamped group.

Selection of patients for their groups is disturbing (if
not despicable):

    "The original plan was to study only infants born
    of ward mothers.  Ward patients were assigned
    in rotation to Service I or II when first seen at the
    hospital...
    ...It became necessary to include private
    patients in the premature group to increase
    enrollment; the planned randomoization of
    these infants as to time of cord clamping failed."
    [1, pp893-4]

In the 1960s health insurance was not provided by
employers, and obstetric care was not normally
covered in any insurance policies.  I was a ward
patient.  My husband was a graduate student, and
income from my work was our primary support with
much disapproval and reprimand from everyone,
including the obstetric residents in the clinic.  In the
1960s women with young children or more than four
to six months pregnant were not supposed to work
[2].  This attitude was very detrimental to the care
received by many of us who could not afford the fees
of private and more experienced doctors.

Taylor et al. stated that their interest was in the
effect of early versus delayed cord-clamping on the
"clinical condition" of the newborn.  Their measures
of clinical condition were respiratory rate and
presence or absence of retractions.  They
concluded from these observations, "that the time of
cord clamping had little effect on neonatal survival."

No deaths occurred among full-term infants in the
study by Taylor et al.  However, in contrast to the
research cited of von Engel (1885) and Bound et al.
(1962) [3, 4], more deaths of premature infants
occurred among those with delayed clamping of the
umbilical cord.  On the other hand, at autopsy
atelectasis and hyaline membrane disease of the
lungs was found more often in premature infants with
early clamping of the cord.

Taylor et al. commented that the disagreement
between their findings and those of von Engel and
Bound et al. was unexplained, but they proceeded to
criticise the experimental design of these two earlier
studies.  They also noted again that they had been
unable to enforce randomization of premature
infants selected for early or delayed clamping of the
cord.

Randomized controlled trials continue to be held in
highest esteem, but this needs to be questioned.  
Understanding of the physiology of fetal to neonatal
transition should guide obstetric practice, not
"evidence" obtained from random treatment of our
children according to one protocol or another.

Taylor et al. cited the editorial in the Lancet [5] that
accompanied the papers by Bound et al. (1962) and
Secher and Karlberg (1962) [4, 6], and noted that
the editorial provided a review of research on
placental transfusion.  The editorial cited a review by
Köstlin (1898) of nearly sixty publications in the
nineteenth century or earlier, plus papers by Budin
(1875), von Engel (1885), Jäykkä (1954), Gunther
(1957), Mahaffey and Rossdale (1959), and an
unpublished version of the paper by Taylor et al.
(1963) [7-11].

Taylor et al. also cited McCausland (1949), who had
surveyed mid-twentieth century obstetric practice
and found that 59 percent of obstetricians still
practiced delayed clamping of the cord and 38
percent stripped blood from the cord into the infant
[12].  Taylor et al. offered the following opinion:

    "Proponents of delayed clamping of the cord
    suggest that the placental transfusion (which
    may exceed 30 percent of the infant's estimated
    blood volume) benefits the infant by filling his
    expanding pulmonary vascular bed, by initiating
    or aiding initial lung expansion, by prevention of
    'hematogenic shock,' and by increasing
    hemoglobin concentration and supply of body
    iron."  [1, p893]

Their view, on the contrary, was that allowing
placental transfusion led to temporary hypertension
in contrast to early-clamped infants for whom no
significant elevation of blood pressure occurred.  
This reflects a belief that placental blood is not part
of the total volume of fetal blood.

The placenta provides oxygen to the fetal circulatory
system.  Pulmonary blood volume is minimal during
fetal life, but after birth no one would suggest that
pulmonary blood is separate from that circulating
through other organs of the infant.  If placental blood
is not transferred to the infant at birth, what will be
the source of blood going to the newly inflated
lungs?  It will have to be drawn from other organs
including the brain.

Is any more experimentation with human subjects
really warranted?

(in progress)
to
References
  1. Taylor PM, Bright NH,
    Birchard EL. Effect of early
    versus delayed clamping of
    the cord on the clinical
    condition of the newborn
    infant.
  2. Friedan, B (1963) The
    Feminine Mystique.
  3. von Engel G (1885) Über
    den Zeitpunkt der
    Abnabelung.
  4. Bound JP et al. (1962)
    Prevention of pulmonary
    syndrome of the newborn.
  5. Anonymous [Lancet editorial]
    (1962). Placental
    Transfusion.
  6. Secher O, Karlberg P (1962)
    Placental blood-transfusion:
    For Newborns delivered by
    Cæsarean Section.
  7. Köstlin R. Ueber das
    Zustandekommen und die
    Bedeutung der postnatalen
    Transfusion.
  8. Budin, P (1875) A quel
    moment doit-on pratiquer la
    ligature du cordon ombilical?
  9. Jäykkä S (1954) A new
    theory concerning the
    mechanism of the initiation
    of respiration in the
    newborn; a preliminary
    report.
  10. Gunther M (1957) The
    transfer of blood between
    baby and placenta in the
    minutes after birth.
  11. Mahaffey LW, Rossdale PD
    (1959) A convulsive
    syndrome in newborn foals
    resembling pulmonary
    syndrome in the newborn
    infant.
  12. McCausland AM et al. (1949)
    Management of cord and
    placental blood and its effect
    upon the newborn; part I.
Full References
  1. Taylor PM, Bright NH, Birchard EL. Effect of early versus delayed clamping
    of the cord on the clinical condition of the newborn infant. Am J Obstet
    Gynecol. 1963 Aug 1;86:893-8.
  2. Friedan B. The Feminine Mystique. New York : Norton, 1963.
  3. von. Engel G (1885) Über den Zeitpunkt der Abnabelung. 1885;
    Centralblatt für Gynäkologie 46: 721-727.
  4. Bound JP, Harvey PW, Bagshaw HB. Prevention of pulmonary syndrome of
    the newborn. Lancet. 1962 Jun 9;1:1200-3.
  5. Anonymous (editorial). Placental Transfusion. Lancet 1962 Jun 9; 279
    (7241):1222-1223.
  6. Secher O, Karlberg P. Placental blood-transfusion: For Newborns
    delivered by Cæsarean Section. Lancet 1962 Jun 9; 279(7241):1203-1205.
  7. Köstlin R. Ueber das Zustandekommen und die Bedeutung der postnatalen
    Transfusion. Zeitschrift für Geburtshülfe und Gynäkologia 1898; 39:98-136.
  8. Budin, P (1875) A quel moment doit-on pratiquer la ligature du cordon
    ombilical?  Progres Medical 3:750-751, 765-767, (1876) 4:2-3.
  9. Jäykkä S. A new theory concerning the mechanism of the initiation of
    respiration in the newborn; a preliminary report. Acta Paediatr. 1954 Sep;
    43(5):399-410.
  10. Gunther M. The transfer of blood between baby and placenta in the
    minutes after birth. Lancet. 1957 Jun 22;272(6982):1277-80.
  11. Mahaffey LW, Rossdale PD. A convulsive syndrome in newborn foals
    resembling pulmonary syndrome in the newborn infant. Lancet. 1959 Jun
    13; 1(7085):1223-5.
  12. McCausland AM, Homes F, Schumann WR. Management of cord and
    placental blood and its effect upon the newborn; part I. California Medicine
    1949;71(3):190-196.
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